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It exists because of a specific and testable hypothesis: that the lipolytic activity attributed to growth hormone resides in a discrete C-terminal domain, and that this domain can be excised from the protein and used on its own without carrying along the growth-promoting, IGF-1-inducing and insulin-antagonizing activities that require the intact molecule and its receptor-binding surfaces

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The problem is that many chronic injuries involve structural deficits that require deeper remodeling than symptom management can provide