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Endothelial BMAL1 decline during aging leads to bone loss by destabilizing extracellular fibrillin-1
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The present study demonstrated that NNMT inhibitor administered during adipocyte differentiation blocked the accumulation of lipid droplets, suggesting suppressed lipogenesis and programed development of mature adipocytes from pre-adipocyte 3T3-L1 precursor cells (i.e., “adipogenesis”)